Overview of Vitiligo Research April - June 2013

This is a review of research results in the vitiligo field which were indexed in the PubMed database (www.ncbi.nlm.nih.gov/pubmed) for the period from April 2013 till June 2013. Abstracts of papers were retrieved from the PubMed database using the search term “vitiligo” with a filter set up to retrieve records with creation date between April 01, 2013 and June 30, 2013. Retrieved records were manually checked for a relevance to and significance for the field of vitiligo research. Fifty six records have been selected for inclusion into this quarterly overview.

HIGHLIGHTS

  • Experimental data suggest that vitiligo might be considered as a degenerative disease
  • MIA protein can be involved in stress-induced melanocyte loss in vitiligo
  • Revealed crucial role of BACH2 in shaping regulatory T-cell response in conjunction with association of BACH2 genetic variations with vitiligo support regulatory T-cell involvement in vitiligo pathogenesis and opens an avenue for exploring mechanisms of regulatory T-cell
  • Two new mouse models of vitiligo have been developed
  • Luteolin may be beneficial in vitiligo treatment
  • Dermal mesenchymal stem cells can help in autologous melanocyte transplantation
  • Dermal fibroblast subpopulation can be used as a source for generation of functional melanocytes
  • Wnt signaling pathway emerges as a target to promote repigmentation from hair follicle
  • Childhood vitiligo relapse rate after successful stopping vitiligo progression with systemic steroids is 34.8% within 1 year thus warranting development of preventive strategies
  • History of atopic disorders might predict extent of vitiligo.

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